This article presents uveitis caused by crystalline and intraocular lenses, highlighting diagnosis and treatment options for better vision and quality of life.
The uveal tract is the main source of blood supply to the eyeball. Causes of uveitis include: infection, tumor, trauma, and autoimmune. Chemical mediators of acute inflammation include serotonin, complement, and plasmin. Leukotrienes, kinins, and prostaglandins modify phase 2 of the acute inflammatory response through antagonism of vasoconstrictor factors. The complex of immunity response is a leukocyte attractant. Polymorphonuclear leukocytes, basophils, and mast cells all contribute to inflammation but lymphocytes predominate in uveitis and endophthalmitis. The International Uveitis Study Group (IUSG) classified uveitis as follows: Anterior uveitis; Intermediate uveitis; Posterior uveitis and Pan uveitis. In this article, uveitis caused by crystalline lens and intraocular lens with or without dislocation is presented including early diagnosis/ treatment for vision maintenance as well as for better quality of life.
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Duong Dieu (2025) studied this question.
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