Case report reveals treatment success of Guillain–Barré syndrome after malaria, highlighting neurological deficits.
Guillain–Barré syndrome (GBS) is an acute immune‐mediated polyradiculoneuropathy often triggered by infections. Malaria, a common parasitic disease in endemic regions, has rarely been associated with GBS. We report a case of a 21‐year‐old male from rural Ethiopia who developed GBS following Plasmodium vivax malaria. The patient presented with ascending paralysis, areflexia, dysphagia, dysarthria, and autonomic dysfunction. Cerebrospinal fluid (CSF) analysis revealed albumin‐cytologic dissociation, which further strengthens the diagnosis of GBS. He was successfully treated with intravenous immunoglobulin (IVIg) and supportive care. The patient achieved full recovery after 8 weeks from the onset of weakness. This case highlights the importance of considering GBS in patients with recent malaria infection and neurological deficits, particularly in malaria‐endemic regions. The rarity of such cases in the literature and the unique clinical course make this report noteworthy.
No takes yet. Share an insight, caveat, or question.
Abdela et al. (2025) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: