Supplementation reduces oxidative damage and apoptosis in human neuronal cells, suggesting therapeutic potential.
BA and Qe, particularly in combination, protect neuronal cells from PQ-induced oxidative and mitochondrial damage by restoring redox balance, stabilizing mitochondria, regulating Ca²⁺ homeostasis and modulating apoptosis and attenuating irritation in the in ovo model. These results suggest that BA and Qe may be promising complementary therapeutics to attenuate neurotoxicity caused by environmental toxins.
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Aşkın Tekin (2025) studied this question.
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