Observational analysis reveals Tanshinone IIA reduces oxidative stress and apoptosis in acute kidney injury, suggesting therapeutic potential.
These findings suggest that Tan IIA protects against LPS-induced AKI by inhibiting RIP3 expression and restoring Nrf2-mediated antioxidant defenses, thereby attenuating oxidative stress and apoptosis.
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Shu Zhang (2025) studied this question.
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