Findings demonstrate tanshinone IIA reduces myocardial injury via MALT1/NF-κB/NLRP3 signaling in heart conditions.
Tan IIA attenuated I/R-induced myocardial inflammation and apoptosis through the inhibition of the MALT1/NF-κB/NLRP3 signaling pathway, ultimately alleviating myocardial I/R injury.
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Yu et al. (2026) studied this question.
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