Key result
Aneurysm coiling followed by Sirolimus DCB successfully treats concurrent subarachnoid hemorrhage and AMI.
Why the study?
The simultaneous occurrence of subarachnoid hemorrhage and acute myocardial infarction presents significant diagnostic and therapeutic challenges requiring multidisciplinary management.
Case Report (n=1)
No
Answers are grounded in this paper and cite their sources.
Demonstrates the feasibility of using a drug-coated balloon and delayed single antiplatelet therapy to manage acute myocardial infarction in a patient with high bleeding risk due to concurrent subarachnoid hemorrhage.
Case RepoRtanticoagulants, antiplatelets, or delay in cardiological as well as neurosurgical intervention.Moreover, the use of a drug-coated balloon (DCB) to keep a balance between minimum risk of intracranial bleeding and prevention of stent thrombosis/stenosis was also a unique approach for this patient with high bleeding risk (HBR).We present a case of SAH complicated by AMI where timely and judicious neurosurgical and cardiological intervention has IntroductIonAcute myocardial infarction (AMI) is a rare but life-threatening association with subarachnoid hemorrhage (SAH). 1 Acute coronary syndrome (ACS), which is a potentially reversible medical emergency, can lead to cardiac arrest if not intervened in a timely manner.ACS can result from cardiac ischemia leading to myocardial injury or necrosis.Various neurological disorders can mimic the electrocardiogram (ECG) changes observed in ACS.These conditions include SAH, intracranial hemorrhage (ICH), subdural hematoma, brain tumors, brain abscesses, tuberculoma, and meningitis.However, these observations are not very common in clinical practice and the literature. 2 Intracranial hemorrhage (trauma-induced or spontaneous) can elicit sympathetic stimulation, leading to cardiac repolarization and thus inducing ST changes on ECG.Raised intracranial tension and activation of the sympathetic nervous system can stimulate catecholamine release, influencing cardiac dynamics.3 However, differentiating primary cardiac pathology from neurologically (SAH) induced ECG alterations involves intricate assessment of clinical symptoms, serial troponin level tracing, and imaging studies to accurately diagnose the underlying cause of abnormal ST segment changes, which is crucial for appropriate clinical management.This was an extremely rare and crucial clinical scenario of diagnostic dilemma, as misdiagnosis can lead to delayed use of 1,
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Basu et al. (2025) conducted a case report in Subarachnoid Hemorrhage and Acute Myocardial Infarction (n=1). Aneurysm coiling and PTCA with Sirolimus drug-coated balloon was evaluated on Clinical recovery. A multidisciplinary approach involving aneurysm coiling followed by percutaneous coronary intervention with a Sirolimus drug-coated balloon successfully treated a patient with concurrent subarachnoid hemorrhage and acute myocardial infarction.
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