Type 1 Diabetes as a Systems Transition — A Universal Resonance Model (URM) Perspective

Type 1 diabetes (T1D) is traditionally described as an autoimmune destruction of pancreatic beta cells, yet its development is prolonged, dynamic, and strongly phase-dependent. This paper presents a conceptual interpretation of T1D as a long-term systems transition from a Universal Resonance Model (URM) perspective. Disease dynamics are described in terms of declining resilience, maladaptive resonance between immune, beta-cell, and metabolic loops, phase shifts from compensatory to insulin-dependent regimes, and eventual structural and functional lock-in. Biomarkers are interpreted as phase markers and instability signals rather than static disease labels, and preventive or therapeutic effects are viewed as inherently timing-critical. Conceptual framework only; no new experimental, clinical, or epidemiological data are included.