Key result
Smoking is linked to altered Tp-e intervals and ratios indicating increased ventricular arrhythmia risk.
Why the study?
Smoking may lead to ventricular arrhythmias and sudden cardiac death, prompting evaluation of the relationship between cigarette smoking and ventricular repolarisation dispersion using novel electrocardiographic parameters.
Does cigarette smoking alter ventricular repolarisation dispersion indices such as Tp-e interval, Tp-e/QT, and Tp-e/QTc ratios?
Does cigarette smoking alter ventricular repolarisation dispersion indices such as Tp-e interval, Tp-e/QT, and Tp-e/QTc ratios?
Smoking history should inform Tp-e/QT interpretation for arrhythmia risk; extends observational data but remains hypothesis-generating pending outcome trials.
Background Smoking may lead to ventricular arrhythmias and sudden cardiac death via altering ventricular recovery time dispersion indices such as QT interval and QT dispersion (QTd). The Tp-e/QT and Tp-e/QTc ratios are also known as predictors of ventricular arrhythmogenesis. The aim of this study was to evaluate the relationship between cigarette smoking and ventricular repolarisation dispersion using these novel electrocardiographic parameters.
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Cakici et al. (2014) studied this question. Smoking is associated with altered Tp-e interval, Tp-e/QT, and Tp-e/QTc ratios, indicating increased ventricular arrhythmia risk.
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