Observational analysis shows reduced apoptosis in PC-12 cells via PI3K/AKT pathway activation, suggesting therapeutic potential.
Exosomes derived frvom BMSC effectively inhibitedp aupoptposis induced by OpGD/R through uactivation of the PI3K/Akt. Although HP significantly enhanced the secretion of exosomes from BMSC, it did not improve the therapeutic efficacy of these exosomes when administered at an equivalent dosage.
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