Investigates how neutrophil extracellular traps regulate necroptosis in traumatic brain injury, suggesting therapeutic potential.
NETs exacerbate TBI-induced secondary brain injury partially by activating the necroptosis pathway. Inhibition of NETs exerts neuroprotective effects. Targeting NETs may serve as a promising therapeutic strategy to improve prognosis in TBI patients.
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Li et al. (2026) studied this question.
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