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March 11, 2026PLoS ONEOpen Access

Expression of concern: HSV-2 regulates monocyte inflammatory response via the Fas/FasL pathway

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Key Points

To investigate how HSV-2 influences the inflammatory response in monocytes via the Fas/FasL pathway.
Analyzing monocyte behavior in response to HSV-2 exposure.
Investigating the role of Fas/FasL in the inflammatory response.
Measuring cytokine levels and immune markers.
HSV-2 significantly alters the inflammatory profile of monocytes.
Fas/FasL pathway activation is implicated in this regulation.
Changes in cytokine levels indicate a shift in immune response.

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Implication

Expression of concern reveals HSV-2 alters inflammatory response in monocytes, suggesting implications for immune system understanding.

Abstract

Expression of concern: HSV-2 regulates

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Cite This Study

The Editors (2026) studied this question.

synapsesocial.com/papers/69b64d48b42794e3e660e0c2 https://doi.org/10.1371/journal.pone.0344636

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Fas/FasL-Mediated Apoptosis and Inflammation Contribute to Recovery from HSV-2-Mediated Spinal Cord Infection2024
2Lack of Fas/FasL Does Not Protect from Latent Herpes Simplex 1 Infection but Decreases Virus-Induced Neurodegeneration2025
3Epithelial interferon-γ responsiveness shapes chemokine induction and clinical outcomes in herpes simplex virus infectionOriginal Research · 2026
4Herpes simplex virus type 1 interferes with signal transducer and activator of transcription 3 expression in infected mature dendritic cellsOriginal Research · 2026
5HSV-1 and Cellular miRNAs in CSF-Derived Exosomes as Diagnostically Relevant Biomarkers for Neuroinflammation2024 · 17 citations

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