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April 3, 2026Journal of Inflammation Research2 citationsOpen Access

The Role of Histone Modifications in Acute Lung Injury: Molecular Mechanisms and Potential of Traditional Chinese Medicine Treatment

ZYZhiqiang YanJWJiayun WangJQJiayi Qiao

Key Points

  • This review examines the role of histone modifications in acute lung injury and evaluates the potential of traditional Chinese medicine for treatment.
  • Review of molecular mechanisms related to histone post-translational modifications
  • Integration of histone acetylation, methylation, lactylation, and citrullination in ALI/ARDS pathology
  • Assessment of therapeutic strategies and the role of traditional Chinese medicine interventions
  • Histone modifications modulate inflammatory responses and amplify lung injury mechanisms
  • Traditional Chinese medicine shows potential to target multiple pathways involved in ALI
  • Dynamic epigenetic biomarkers, such as serum CitH3, may enable personalized therapy approaches

Abstract

Background: Acute lung injury (ALI) and acute respiratory distress syndrome (ARDS) are life-threatening conditions characterized by excessive inflammation, alveolar-capillary barrier disruption, and immunothrombosis. Mortality remains persistently high due to the lack of effective targeted therapies. Emerging evidence indicates that histone post-translational modifications (PTMs) play dual regulatory roles by modulating intranuclear gene transcription and, upon extracellular release, amplifying damage-associated molecular pattern (DAMP) signaling. Scope: This review proposes a unified pathological framework termed the “Nuclear Epigenetic Remodeling‒Extracellular DAMP Amplification Circuit.” By conceptualizing this as an inside-out pathological cascade, we integrate four principal histone modifications—acetylation, methylation, lactylation, and citrullination—and evaluate three-tiered therapeutic strategies alongside the unique multi-target potential of traditional Chinese medicine (TCM). Key Findings: Mechanistically, intranuclear HDAC3/6 drive inflammatory gene expression and pyroptosis, while cytoprotective SIRT1/3/6 suppress inflammasome activation and ferroptosis; concurrently, histone lactylation at H3K18 and H3K14 bridges glycolytic metabolism with ferroptosis and glycocalyx degradation; ultimately, externalized citrullinated histone H3 (CitH3) propagates a thrombo-inflammatory cascade driving NET-mediated immunothrombosis. Corresponding to this cascade, TCM-derived interventions collectively show promising potential to act across all therapeutic tiers by modulating the HDAC/SIRT acetylation axis, suppressing the PAD4–CitH3–NETs cascade, and regulating lactylation-linked metabolic pathways. Conclusion: We propose a precision medicine approach integrating dynamic epigenetic biomarkers (eg, serum CitH3 and H3K18la) with a multi-tiered intervention framework. This paradigm aims to shift ALI/ARDS treatment from empirical supportive care toward mechanism-based, individualized targeted therapies. Keywords: acute lung injury, acute respiratory distress syndrome, histone modifications, extracellular histones, neutrophil extracellular traps, immune-related thrombosis, traditional Chinese medicine

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Cite This Study

Yan et al. (2026) studied this question.

synapsesocial.com/papers/69cf59315a333a8214609dd2https://doi.org/10.2147/jir.s588149
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