What question did this study set out to answer?

This research aims to clarify how ligand-induced ubiquitination affects the ERECTA receptor kinases and their role in stomatal development.

April 6, 2026New PhytologistOpen Access

Ligand‐induced ubiquitination regulates endocytosis and homeostasis of the ERECTA receptor kinase for stomatal development

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This research aims to clarify how ligand-induced ubiquitination affects the ERECTA receptor kinases and their role in stomatal development.
Performed ubiquitome profiling to identify ubiquitination sites on ERECTA
Conducted site-directed mutagenesis to create ERECTA 3KR mutant
Analyzed phenotypes of ERECTA and ERECTA 3KR in stomatal and pedicel development
Used biochemical assays to study ligand-induced ubiquitination processes
Ligand-induced ubiquitination was shown to regulate ERECTA endocytosis and degradation
ERECTA 3KR mutant displayed hyperactivity leading to excessive stomatal inhibition
Poor co-localization with endocytic markers in ERECTA 3KR indicated impaired endocytosis
Higher levels of ERECTA 3KR protein were observed compared to wild-type, reflecting reduced sensitivity to ligands

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Implication

Research reveals that ligand-induced ubiquitination affects ERECTA receptor endocytosis in Arabidopsis, suggesting implications for stomatal development.

Cite This Study

Chen et al. (2026) studied this question.

synapsesocial.com/papers/69d34e579c07852e0af97e08 https://doi.org/10.1111/nph.71151

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Implication

Research reveals that ligand-induced ubiquitination affects ERECTA receptor endocytosis in Arabidopsis, suggesting implications for stomatal development.

Ligand‐induced ubiquitination regulates endocytosis and homeostasis of the ERECTA receptor kinase for stomatal development

Key Points

Discussion

Implication

Cite This Study

Ligand‐induced ubiquitination regulates endocytosis and homeostasis of the ERECTA receptor kinase for stomatal development

Key Points

Discussion

Implication

Cite This Study

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