Answers are grounded in this paper and cite their sources.
These findings show for the first time to our knowledge that reactive oxygen species directly link HIF-1alpha and NF-kappaB, implicating an important pathophysiological role of this novel pathway in disorders associated with elevated levels of reactive oxygen species.
No takes yet. Share an insight, caveat, or question.
Bonello et al. (2007) studied this question.
Synapse has enriched 10 related papers. Consider them for comparative context: