Enhanced parasympathetic neural activity lowers exercise heart rate at high altitude, while hypoxia mediates the attenuation of peak exercise cardiac output.
Should not yet change clinical practice for exercise at altitude; extends understanding of autonomic HR control in hypoxia but remains hypothesis-generating.
Enhanced parasympathetic neural activity accounts for the lowering of HR during exercise at ALT without influencing Q. The abrupt restoration of peak exercise Q in chronic hypoxia to maximal SL values when arterial PO(2) and SO(2) are similarly increased suggests hypoxia-mediated attenuation of Q.
No takes yet. Share an insight, caveat, or question.
Boushel et al. (2001) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: