Answers are grounded in this paper and cite their sources.
The results suggest the existence of a novel molecular mechanism by which astrocytes transform glutamatergic excitation into GABAergic inhibition providing an adjustable, in situ negative feedback on the excitability of neurons.
No takes yet. Share an insight, caveat, or question.
Héja et al. (2012) studied this question.
Synapse has enriched 10 related papers. Consider them for comparative context: