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Reveals how neutrophil extracellular traps drive organ injury in complement-mediated thrombotic microangiopathy, indicating a need for targeted therapies.
Levels of NETs biomarkers increased in complement-mediated TMA and drove multi-organ injury by shedding CD59 from HRGECs to enhance complement activation and participate in coagulation activation.
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Liu et al. (2026) studied this question.
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