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October 29, 1996Proceedings of the National Academy of SciencesOpen Access

Arrested development of embryonic red cell precursors in mouse embryos lacking transcription factor GATA-1.

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Authors

YFYuko FujiwaraBoston Children's HospitalCBCarol BrowneHoward Hughes Medical InstituteKCK. C. CunniffBrigham and Women's Hospital

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Cite This Study

Fujiwara et al. (1996) studied this question.

synapsesocial.com/papers/69d8d3f95c3030ff03d1a8ebhttps://doi.org/10.1073/pnas.93.22.12355
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Transcription factor GATA-1 permits survival and maturation of erythroid precursors by preventing apoptosis.1995 · 305 citations
  2. 2Hematopoietic Commitment During Embryonic Stem Cell Differentiation in Culture1993 · 267 citations
  3. 3The major human erythroid DNA-binding protein (GF-1): primary sequence and localization of the gene to the X chromosome.1990 · 153 citations
  4. 4Novel insights into erythroid development revealed through in vitro differentiation of GATA-1 embryonic stem cells.1994 · 565 citations
  5. 5Regulation of Globin Gene Expression in Erythroid Cells1995 · 137 citations