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Reveals elevated inflammation and blood clotting markers in frail older adults without diabetes or cardiovascular disease, indicating a distinct physiological basis for frailty.
These findings support the hypothesis that there is a specific physiological basis to the geriatric syndrome of frailty that is characterized in part by increased inflammation and elevated markers of blood clotting and that these physiological differences persist when those with diabetes and cardiovascular disease are excluded.
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Jeremy Walston (2002) studied this question.
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