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February 28, 2014DiabetologiaOpen Access

Canagliflozin, a sodium glucose co-transporter 2 inhibitor, improves model-based indices of beta cell function in patients with type 2 diabetes

David Polidori Andrea Mari Ele Ferrannini

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Clinicaltrials.gov NCT01081834 (Study 1); NCT01106625 (Study 2); NCT01137812 (Study 3).

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Polidori et al. (2014) studied this question.

synapsesocial.com/papers/69d9ae102a25b240b7a3d732 https://doi.org/10.1007/s00125-014-3196-x

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1β-Cell Function in Subjects Spanning the Range from Normal Glucose Tolerance to Overt Diabetes: A New AnalysisCross-Sectional Study · 2005 · 542 citations
2Primary care physicians and insulin initiation: multiple barriers, lack of knowledge or both?Original Research · 2008 · 44 citations
3Management of Hyperglycemia in Type 2 Diabetes: A Patient-Centered ApproachOriginal Research · 2012 · 3,212 citations
4Canagliflozin Lowers Postprandial Glucose and Insulin by Delaying Intestinal Glucose Absorption in Addition to Increasing Urinary Glucose ExcretionOriginal Research · 2013 · 265 citations
5Validation of a Novel Method for Determining the Renal Threshold for Glucose Excretion in Untreated and Canagliflozin-treated Subjects With Type 2 Diabetes MellitusOriginal Research · 2013 · 87 citations

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Effect of Canagliflozin on Renal Threshold for Glucose, Glycemia, and Body Weight in Normal and Diabetic Animal Models2012

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