Why the study?
Cardiac arrhythmias are a significant cause of global mortality, and animal models are essential tools to study complex cellular and whole-heart arrhythmogenic mechanisms and test therapeutic interventions.
This review highlights the utility of animal models in studying the molecular, cellular, and whole-heart mechanisms of cardiac arrhythmias and testing therapeutic interventions.
Informs preclinical arrhythmia research design; leaves open direct clinical translation of model findings.
Cardiac arrhythmias are a significant cause of morbidity and mortality worldwide, accounting for 10% to 15% of all deaths. Although most arrhythmias are due to acquired heart disease, inherited channelopathies and cardiomyopathies disproportionately affect children and young adults. Arrhythmogenesis is complex, involving anatomic structure, ion channels and regulatory proteins, and the interplay between cells in the conduction system, cardiomyocytes, fibroblasts, and the immune system. Animal models of arrhythmia are powerful tools for studying not only molecular and cellular mechanism of arrhythmogenesis but also more complex mechanisms at the whole heart level, and for testing therapeutic interventions. This review summarizes basic and clinical arrhythmia mechanisms followed by an in-depth review of published animal models of genetic and acquired arrhythmia disorders.
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Blackwell et al. (2022) studied this question.
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