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February 5, 2009BloodOpen Access

Molecular mimicry of host sialylated glycans allows a bacterial pathogen to engage neutrophil Siglec-9 and dampen the innate immune response

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Authors

ACAaron F. CarlinUniversity of California, San DiegoSUSatoshi UchiyamaNational Cancer Center Hospital EastYCYung‐Chi ChangNational Taiwan University

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Carlin et al. (2009) studied this question.

synapsesocial.com/papers/69da3ecc0d540cafc5838d33https://doi.org/10.1182/blood-2008-11-187302
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Cryptic sialic acid binding lectins on human blood leukocytes can be unmasked by sialidase treatment or cellular activation1999 · 99 citations
  2. 2Growth of Male-Specific Bacteriophage in Proteus mirabilis Harboring F-Genotes Derived from Escherichia coli1965 · 24 citations
  3. 3Siglec-9 transduces apoptotic and nonapoptotic death signals into neutrophils depending on the proinflammatory cytokine environment2005 · 219 citations
  4. 4NeuA Sialic Acid O-Acetylesterase Activity Modulates O-Acetylation of Capsular Polysaccharide in Group B Streptococcus2007 · 50 citations
  5. 5Siglecs—the major subfamily of I-type lectins2005 · 524 citations