Loss of perivascular aquaporin-4 localization impairs glymphatic exchange and promotes amyloid β plaque formation in mice

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These findings suggest that the loss of perivascular AQP4 localization may contribute to the development of AD pathology in human populations.

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Simon et al. (2022) studied this question.

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These findings suggest that the loss of perivascular AQP4 localization may contribute to the development of AD pathology in human populations.