The research aims to explore the potential liver toxicity of camphor and its underlying mechanisms.
Utilized network pharmacology to analyze multiple molecular targets.
Employed molecular docking techniques to investigate interactions.
Focused on zebrafish larvae as a model organism.
Identified potential hepatotoxic effects of camphor in zebrafish.
Highlighted involvement of multiple biological targets.
Primary targets identified include GSR, GCLC, and NQO1.
Abstract
This study has identified potential hepatotoxicity of camphor in zebrafish larvae, with its toxic mechanism involving multiple targets and pathways. The primary targets are likely GSR, GCLC and NQO1.