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July 1, 2003Journal of Biological ChemistryOpen Access

Glycosylphosphatidylinositol-anchored Ceruloplasmin Is Required for Iron Efflux from Cells in the Central Nervous System

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Authors

SJSuh Young JeongOregon Health & Science UniversitySDSamuel DavidDüsseldorf University Hospital

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Jeong et al. (2003) studied this question.

synapsesocial.com/papers/69dbdfb9e6ab964fb083713fhttps://doi.org/10.1074/jbc.m301988200
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1MIP-1alpha, MCP-1, GM-CSF, and TNF-alpha control the immune cell response that mediates rapid phagocytosis of myelin from the adult mouse spinal cord.2001 · 150 citations
  2. 2Ceruloplasmin Regulates Iron Levels in the CNS and Prevents Free Radical Injury2002 · 374 citations
  3. 3The Possible Significance of the Ferrous Oxidase Activity of Ceruloplasmin in Normal Human Serum1966 · 677 citations
  4. 4Control of Transferrin Receptor Expression via Nitric Oxide-mediated Modulation of Iron-regulatory Protein 21999 · 92 citations
  5. 5Expression of the ceruloplasmin gene in the human retina and brain: implications for a pathogenic model in aceruloplasminemia1996 · 162 citations