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April 1, 2001DiabetesOpen Access

Differential Splicing of the IA-2 mRNA in Pancreas and Lymphoid Organs as a Permissive Genetic Mechanism for Autoimmunity Against the IA-2 Type 1 Diabetes Autoantigen

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Authors

JDJuan DíezYPYongsoo ParkMZMarkus Zeller

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Díez et al. (2001) studied this question.

synapsesocial.com/papers/69dd1ca84006ebb8efe530d9https://doi.org/10.2337/diabetes.50.4.895
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Identification of protein tyrosine phosphatase-like IA2 (islet cell antigen 512) as the insulin-dependent diabetes-related 37/40K autoantigen and a target of islet-cell antibodies1995 · 241 citations
  2. 2Islet cell antigen 512 is a diabetes-specific islet autoantigen related to protein tyrosine phosphatases.1994 · 305 citations
  3. 3IA-2 (Islet Cell Antigen 512) Is the Primary Target of Humoral Autoimmunity Against Type 1 Diabetes-Associated Tyrosine Phosphatase Autoantigens1998 · 83 citations
  4. 4Post‐translational modifications of ICA512, a receptor tyrosine phosphatase‐like protein of secretory granules1999 · 53 citations
  5. 5Early Development and Spreading of Autoantibodies to Epitopes of IA-2 and Their Association with Progression to Type 1 Diabetes1998 · 100 citations