January 30, 2013The Journal of Clinical Endocrinology & MetabolismOpen Access

DNA Mismatch Repair Protein (MSH6) Correlated With the Responses of Atypical Pituitary Adenomas and Pituitary Carcinomas to Temozolomide: The National Cooperative Study by the Japan Society for Hypothalamic and Pituitary Tumors

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THToshio HIROHATATeikyo University KAKenichiro AsanoKurashiki Central Hospital YOYoshikazu OgawaHigashi Osaka City General Hospital

Abstract

This study showed that preserving MSH6 function was contributory to the effectiveness of TMZ in malignant pituitary neoplasms. It is necessary to survey more cases and evaluate multifactor analyses.

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Hirohata et al. (2013) studied this question.

synapsesocial.com/papers/69de882f6bae133e7de93b7a https://doi.org/10.1210/jc.2012-2924

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Abstract

This study showed that preserving MSH6 function was contributory to the effectiveness of TMZ in malignant pituitary neoplasms. It is necessary to survey more cases and evaluate multifactor analyses.

DNA Mismatch Repair Protein (MSH6) Correlated With the Responses of Atypical Pituitary Adenomas and Pituitary Carcinomas to Temozolomide: The National Cooperative Study by the Japan Society for Hypothalamic and Pituitary Tumors

Abstract

Discussion

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Cite This Study

DNA Mismatch Repair Protein (MSH6) Correlated With the Responses of Atypical Pituitary Adenomas and Pituitary Carcinomas to Temozolomide: The National Cooperative Study by the Japan Society for Hypothalamic and Pituitary Tumors

Abstract

Discussion

Member takes

Cite This Study

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