Beneficial Role of Erythrocyte Adenosine A2B Receptor–Mediated AMP-Activated Protein Kinase Activation in High-Altitude Hypoxia

Hong Liu; Yujin Zhang; Hongyu Wu; Angelo D’Alessandro; Gennady G. Yegutkin; Anren Song; Kaiqi Sun; Jessica Li; Ning-Yuan Cheng; Aji Huang; Y. Edward Wen; Ting Weng; Fayong Luo; Travis Nemkov; Hong Sun; Rodney E. Kellems; Harry Karmouty‐Quintana; Kirk C. Hansen; Bihong Zhao; Andrew W. Subudhi; Sonja Jameson-Van Houten; Colleen G. Julian; Andrew T. Lovering; Holger K. Eltzschig; Michael R. Blackburn; Robert C. Roach; Yang Xia

doi:10.1161/circulationaha.116.021311

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Abstract

Together, human and mouse studies reveal novel mechanisms of hypoxia adaptation and potential therapeutic approaches for counteracting hypoxia-induced tissue damage.

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Liu et al. (2016) studied this question.

synapsesocial.com/papers/69df1588b8d7e945666146d6 https://doi.org/10.1161/circulationaha.116.021311

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Beneficial Role of Erythrocyte Adenosine A2B Receptor–Mediated AMP-Activated Protein Kinase Activation in High-Altitude Hypoxia

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Abstract

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Beneficial Role of Erythrocyte Adenosine A2B Receptor–Mediated AMP-Activated Protein Kinase Activation in High-Altitude Hypoxia

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Abstract

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Cite This Study

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