Study demonstrates hydrogen sulphide reduces reperfusion injury in brain tissue by inhibiting inflammasome activity, suggesting potential therapeutic benefits.
This study demonstrated that exogenous administration of H₂S exerted neuroprotective effects by regulating the Nrf2/HO-1 antioxidant pathway and inhibiting NLRP3 inflammasome activation.
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Hua et al. (2026) studied this question.
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