This article argues that insulin resistance stems from glycogen overload, suggesting a shift in understanding diabetes.
Type 2 diabetes is explained in the conventional model as a state of insulin resistancewith receptor defects, signaling pathway defects, and inflammation. This article argues thatinsulin resistance is not a signaling defect but a logistical problem: the glycogen storesare full. Insulin is a post-it note, glucose is distilled sunlight, and glycans are the body’slanguage. When one cuts away, the need for ad-hoc hypotheses disappears. Falsification issimple: fasting followed by a ketogenic diet normalizes all parameters. Type 2 diabetes isonly a chronic disease if one makes it so.
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Lars H. Hasselby (2026) studied this question.
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