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Demonstrates how estrogen deprivation affects cardiovascular health after pregnancy, suggesting a critical role for estrogen in offspring development.
Prolonged gestational E₂ deprivation programs a reduction in microvascular flow without altering cardiac function; maternal E₂ prevents this, supporting E₂'s role in optimizing postnatal perfusion and metabolic health.
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Turan et al. (2026) studied this question.
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