Key result
Myocardial bridging often remains asymptomatic but links to exertional angina, ACS, arrhythmias, and sudden death.
Why the study?
Although often asymptomatic, myocardial bridges may cause exertional angina, acute coronary syndromes, arrhythmias, syncope, or sudden cardiac death, requiring a review of current pathophysiology, diagnosis, and treatment.
Design
Review
Authors
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Captured external expert commentary on this paper, strongest first. Original sources are linked where available.
“Our understanding of this condition has certainly evolved from the days of assuming most cases to be benign, but now that we have more accurate tests and treatments, we need larger-scale data to guide clinicians on when and how to treat.”
“A good number of people are born with this condition, and yet many of them never present with symptoms, and we only find out incidentally or after death. For those who do present with symptoms, we should perform both anatomic and functional testing to determine whether they are a fit for surgery.”
“However, in certain circumstances, extended compression beyond the systolic phase may indeed impair blood flow through the affected coronary segment, and may lead to various signs of myocardial ischemia such as recurrent chest pain, conduction abnormalities, or rhythm disorders.”
Management of myocardial bridging should remain individualized; this review leaves open the need for prospective outcome data.
This review summarizes the pathophysiology, diagnostic modalities, and therapeutic options for myocardial bridging.
Corban et al. (2014) conducted a review in Myocardial bridging. Myocardial bridging was evaluated. Myocardial bridging is an often asymptomatic anomaly that may be associated with exertional angina, acute coronary syndromes, cardiac arrhythmias, syncope, or sudden cardiac death.
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