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Identifies thalamic serotonin 2A receptors' role in modulating absence seizures, suggesting new therapeutic targets.
OBJECTIVE: R activation depends on thalamocortical (TC) neurons or astrocytes. METHODS: Rs in either TC neurons or astrocytes in the VB. RESULTS: Rs in TC neurons, but not in astrocytes. SIGNIFICANCE: Rs drive seizure modulation, identifying a potential therapeutic target.
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Morais et al. (2026) studied this question.
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