Observational study reveals impaired pulmonary perfusion and hypercoagulability in severe COVID-19 pneumonia, highlighting pulmonary angiopathy and thrombosis as key vascular drivers.
Physiologic, hematologic, and imaging data show not only the presence of a hypercoagulable phenotype in severe COVID-19 pneumonia but also markedly impaired pulmonary perfusion likely caused by pulmonary angiopathy and thrombosis.
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Patel et al. (2020) studied this question.
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