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Preclinical study reveals metabolic pathways governing postpartum diabetes outcomes in rats, highlighting protective lipid signatures during disease resolution.
Specific metabolomic/lipidomic features are associated with GDM postpartum outcomes. GDM-pregnant dams exhibit partial fatty acid oxidation and boosted gluconeogenesis. Resolution of postpartum IGT relies on nervonic acid-sphingomyelin, a β-cell protector. Postpartum IGT persistence suggests muscle insulin resistance and liver dysfunction.
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Paul et al. (2025) studied this question.
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