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Observational study reveals endothelial glycocalyx damage and vascular dysfunction from prolonged door-to-balloon time in STEMI patients, highlighting the critical need for rapid reperfusion.
< 0.01). An increased D2B led to severe eGC shedding, with endothelial dysfunction in a temporal context. eGC components and pro-inflammatory mediators correlated with a prolonged D2B, indicating a time-dependent immune reaction during STEMI, with a decreased NO concentration. Thus, D2B is a crucial factor for eGC damage during STEMI. Clinical evaluation of the eGC condition might serve as an important predictor for the endothelial function of STEMI patients in the future.
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Vahldieck et al. (2023) studied this question.
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