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BACKGROUND: A modification plays an important role in lung adenocarcinoma (LUAD) progression; however, the underlying mechanisms remain unclear. METHODS: A modification on LUAD development. RESULTS: A modification and expression. Furthermore, FBXW7 overexpression in METTL3-depleted cells partially restored LUAD cell suppression in vitro and in vivo. CONCLUSIONS: A-modified FBXW7, thus providing insight into its epigenetic regulatory mechanisms in LUAD initiation and development.
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Wu et al. (2021) studied this question.
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