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Results demonstrate relationships between metabolites and immune responses in toxoplasmosis, suggesting broader implications for infection and inflammation.
. Results revealed relationships between differential metabolite enrichment and variables, including inflammatory signals, pathogen burden, and commensal microbial communities. These data will inform hypotheses about the signals driving specific metabolic adaptation in acute and chronic protozoan infection, with broader implications for infection and inflammation in general.
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Lesani et al. (2025) studied this question.
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