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March 9, 2023Frontiers in PhysiologyOpen Access

Review highlights TRP, Piezo1, and CRAC ion channels as potential therapeutic targets for cardiac fibrosis.

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Why the study?

Persistent cardiac fibrosis leads to heart failure, but an effective treatment strategy has not yet been established.

Design

Review

Key result

This review summarizes the role of various ion channels, including TRP, Piezo1, and CRAC, in myocardial fibroblast activation and cardiac fibrosis to identify potential new therapeutic targets.

Authors

CXChenxv XingLBLimeng BaoWLWeidong Li

Discussion

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Member takes

Overview

May support ion channel targeting to limit fibrosis; hypothesis-generating pending clinical trials.

Structured PICO

P
Population
Cardiac fibroblasts and myofibroblasts in the context of myocardial fibrosis
I
Intervention
Modulation of ion channels (TRP channels, Piezo1, CRAC channels, VGCCs, sodium channels, and potassium channels)
O
Outcome
Myocardial fibrosis (myofibroblast proliferation, contraction, and secretory function)

Ion channels in cardiac fibroblasts play a critical role in the development of myocardial fibrosis and may serve as novel therapeutic targets to prevent heart failure.

Cite This Study

Xing et al. (2023) conducted a review in Cardiac fibrosis. Ion channels (TRP, Piezo1, CRAC, VGCCs, sodium, potassium) was evaluated. This review summarizes the role of various ion channels, including TRP, Piezo1, and CRAC, in myocardial fibroblast activation and cardiac fibrosis to identify potential new therapeutic targets.

synapsesocial.com/papers/6a0a14710e219f8cdd346c27https://doi.org/10.3389/fphys.2023.1138306
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