Key result
Standard medical therapies manage MI primarily caused by oxidative stress and atherosclerosis.
This article provides a general narrative review of the pathophysiology, classification, risk factors, and diagnostic criteria of myocardial infarction.
Reinforces standard MI management in practice; leaves open prospective trials on oxidative stress modulation.
Myocardial infarction (MI), commonly known as a heart attack is the disease of the blood vessels supplying the heart muscle (Myocardium) i.e. coronary heart disease. The area of heart muscle that has either zero flow or so little flow that it cannot sustain cardiac muscle function is said to be infracted and the overall process is called a myocardial infarction. MI are of two types; transmural and subendocardial. Mainly it is caused due to oxidative stress and atherosclerosis.Chest pain is the most common symptom of acute MI and is often described as a sensation of tightness, pressure, or squeezing. Other symptoms include diaphoresis (an excessive form of sweating), Shortness of breath (dyspnea), weakness, light-headedness, nausea, vomiting, and palpitations. The most common symptoms of MI in women include dyspnea, weakness, and fatigue, sleep disturbances. It can be treated by using blockers, diuretics, ACE inhibitors, calcium channel blockers and nitrates.
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Singh et al. (2022) conducted a review in Myocardial Infarction. Myocardial infarction is a cardiovascular disease primarily caused by oxidative stress and atherosclerosis, which can be managed with beta-blockers, diuretics, ACE inhibitors, calcium channel blockers, and nitrates.
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