Key result
Sustained monomorphic ventricular tachycardia features an excitable gap composing 15% to 45% of the tachycardia cycle length, providing insights for pharmacologic mechanisms and ablation strategies.
Understanding the electrophysiologic properties of postinfarction VT, such as the excitable gap and resetting responses, is crucial for directing ablation and pharmacologic therapy.
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May refine ablation and pharmacologic targeting in reentrant VT; leaves open prospective validation of gap-based strategies.
Richardson et al. (1999) conducted a review in Postinfarction Ventricular Tachycardia. Sustained monomorphic ventricular tachycardia features an excitable gap composing 15% to 45% of the tachycardia cycle length, providing insights for pharmacologic mechanisms and ablation strategies.
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