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September 14, 1999Proceedings of the National Academy of SciencesOpen Access

Prevention of glucose toxicity in HIT-T15 cells and Zucker diabetic fatty rats by antioxidants

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Authors

YTYasuhito TanakaKumamoto University HospitalCGCatherine E. GleasonSunesis (United States)PTPhuong Oanh T. TranPacific Northwest Diabetes Research Institute

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Tanaka et al. (1999) studied this question.

synapsesocial.com/papers/6a0ff2d7d8c5cf602efd67ebhttps://doi.org/10.1073/pnas.96.19.10857
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Also Consider

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  1. 1Differentiation of glucose toxicity from beta cell exhaustion during the evolution of defective insulin gene expression in the pancreatic islet cell line, HIT-T15.1997 · 134 citations
  2. 2Oral Administration of the Antioxidant, N-Acetylcysteine, Abrogates Diabetes-Induced Endothelial Dysfunction1998 · 86 citations
  3. 3Hyperglycemia causes oxidative stress in pancreatic beta-cells of GK rats, a model of type 2 diabetes.1999 · 512 citations
  4. 4Evolution of β-Cell Dysfunction in the Male Zucker Diabetic Fatty Rat1995 · 281 citations
  5. 5Superoxide production from nonenzymatically glycated protein1988 · 304 citations