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August 1, 1996The Journal of Experimental MedicineOpen Access

CTLA-4: a negative regulator of autoimmune disease.

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Authors

NKNitin J. KarandikarCVCarol L. VanderlugtTWTheresa L. Walunas

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Cite This Study

Karandikar et al. (1996) studied this question.

synapsesocial.com/papers/6a0ff617600bce7eadfcad68https://doi.org/10.1084/jem.184.2.783
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Immunosuppression in Vivo by a Soluble Form of the CTLA-4 T Cell Activation Molecule1992 · 807 citations
  2. 2Clonal Expansion Versus Functional Clonal Inactivation: A Costimulatory Signalling Pathway Determines the Outcome of T Cell Antigen Receptor Occupancy1989 · 1,499 citations
  3. 3Functional evidence for epitope spreading in the relapsing pathology of experimental autoimmune encephalomyelitis.1995 · 523 citations
  4. 4Long-Term Survival of Xenogeneic Pancreatic Islet Grafts Induced by CTLA4lg1992 · 1,132 citations
  5. 5CTLA-4 ligation blocks CD28-dependent T cell activation.1996 · 806 citations