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Does not yet support changing metabolic syndrome management; leaves open whether targeting angiotensin-independent renin effects improves outcomes.
The renin angiotensin system (RAS) is important for fluid and blood pressure regulation. Recent studies suggest that an overactive RAS is involved in the metabolic syndrome. This article discusses recent advances on how genetic alteration of the RAS affects cardiovascular and metabolic phenotypes, with a special emphasis on the potential role of angiotensin-independent effects of renin.
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Wang et al. (2010) studied this question.
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