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January 1, 1993Journal of Clinical InvestigationOpen Access

Adverse vascular effects of homocysteine are modulated by endothelium-derived relaxing factor and related oxides of nitrogen.

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Authors

Jonathan S. Stamler
Jonathan S. StamlerGeneral Cardiology
JOJohn A. OsborneEuropean Organization for Nuclear ResearchOJO JarakiBrigham and Women's Hospital

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Stamler et al. (1993) studied this question.

synapsesocial.com/papers/6a10ea25ed67694fb09f9825https://doi.org/10.1172/jci116187
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Radioenzymic determination of homocysteine in plasma and urine.1985 · 307 citations
  2. 2Endothelial cell injury due to copper-catalyzed hydrogen peroxide generation from homocysteine.1986 · 827 citations
  3. 3Hyperhomocysteinemia: An Independent Risk Factor for Vascular Disease1991 · 2,177 citations
  4. 4N-acetylcysteine potentiates platelet inhibition by endothelium-derived relaxing factor.1989 · 249 citations
  5. 5Amino Acids and Collagen-Induced Platelet Aggregation1975 · 9 citations