Key result
Short-term cardiovascular oscillations derive from complex autonomic and haemodynamic inputs, indicating no single mechanism is responsible for respiratory and Mayer wave fluctuations.
This review emphasizes the need for more rigorous experimental design and quantitative modeling to fully understand the complex mechanisms underlying short-term cardiovascular oscillations.
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Should not yet change clinical HRV assessment; challenges baroreflex primacy and leaves open integrated modeling research.
Cohen et al. (2002) conducted a review in Cardiovascular oscillations. Short-term cardiovascular oscillations derive from complex autonomic and haemodynamic inputs, indicating no single mechanism is responsible for respiratory and Mayer wave fluctuations.
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