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June 16, 1997The Journal of Experimental MedicineOpen Access

Tumor Necrosis Factor α and Lymphotoxin α Are Not Required for Induction of Acute Experimental Autoimmune Encephalomyelitis

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KFKarl FreiUniversity of California, RiversideHEHans‐Pietro EugsterUniversity Hospital of ZurichMBMartin BopstRoche (Switzerland)

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Frei et al. (1997) studied this question.

synapsesocial.com/papers/6a12ae5f4891eb3ecca41db6https://doi.org/10.1084/jem.185.12.2177
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Tumor necrosis factor mediates myelin and oligodendrocyte damage in vitro1988 · 1,378 citations
  2. 2Mice with a disrupted IFN-γ gene are susceptible to the induction of experimental autoimmune encephalomyelitis (EAE)1996 · 995 citations
  3. 3Multiple immune abnormalities in tumor necrosis factor and lymphotoxin-α double-deficient mice1996 · 204 citations
  4. 4IFN-gamma plays a critical down-regulatory role in the induction and effector phase of myelin oligodendrocyte glycoprotein-induced autoimmune encephalomyelitis1996 · 638 citations
  5. 5Experimental Autoimmune Encephalomyelitis Induction in Genetically B Cell–deficient Mice1996 · 700 citations