Acute myeloid leukemia (AML) is a genetically heterogeneous clonal disorder characterized by the accumulation of acquired somatic genetic alterations in hematopoietic progenitor cells that alter normal mechanisms of self-renewal, proliferation and differentiation.[1][1] Non-random clonal chromosome
No takes yet. Share an insight, caveat, or question.
Döhner et al. (2008) studied this question.