Key result
MI and osteoporotic fracture are linked by shared systemic inflammation and sympathetic nervous system pathways.
Why the study?
Although epidemiological evidence links myocardial infarction and osteoporotic fracture, the systemic effects and potential damaging bone-heart cross talk between these events remain poorly understood.
This review highlights the bidirectional relationship and shared pathophysiological mechanisms between myocardial infarction and osteoporotic fractures.
Myocardial infarction (MI) and osteoporotic fracture (Fx) are two of the leading causes of mortality and morbidity worldwide. Although these traumatic injuries are treated as if they are independent, there is epidemiological evidence linking the incidence of Fx and MI, thus raising the question of whether each of these events can actively influence the risk of the other. Atherosclerotic cardiovascular disease and osteoporosis, the chronic conditions leading to MI and Fx, are known to have shared pathoetiology. Furthermore, sustained systemic inflammation after traumas such as MI and Fx has been shown to exacerbate both underlying chronic conditions. However, the effects of MI and Fx outside their own system have not been well studied. The sympathetic nervous system (SNS) and the complement system initiate a systemic response after MI that could lead to subsequent changes in bone remodeling through osteoclasts. Similarly, SNS and complement system activation following fracture could lead to heart tissue damage and exacerbate atherosclerosis. To determine whether damaging bone-heart cross talk may be important comorbidity following Fx or MI, this review details the current understanding of bone loss after MI, cardiovascular damage after Fx, and possible shared underlying mechanisms of these processes.
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Tjandra et al. (2024) conducted a review in Myocardial infarction and osteoporotic fracture. Myocardial infarction and osteoporotic fracture are linked by shared pathophysiological mechanisms, including systemic inflammation mediated by the sympathetic nervous system and complement system.
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