Why the study?
Does a bladder-type anti-G suit improve cardiovascular dynamics during +Gz acceleration in lightly anesthetized dogs?
Does a bladder-type anti-G suit improve cardiovascular dynamics during +Gz acceleration in lightly anesthetized dogs?
The anti-G suit maintains perfusion pressure during high sustained G stress but is accompanied by potentially detrimental effects such as elevated central venous pressure and depressed heart rate.
Anti-G suit may preserve pressure in +Gz models but risks elevated LVEDP and bradycardia; leaves open human translation.
Lightly anesthetized dogs underwent 1-min exposure to +Gz acceleration without and with a bladder-type anti-G suit. Prior chronic instrumentation permitted through evaluation of cardiac dynamics. During +3 Gz acceleration all recorded dynamic variables were lowered and transient tachycardia occurred. After acceleration ceases, all pressures and dP/dt exceeded control levels. Inflation of the anti-G suit during +3 Gz eliminated the dramatic effects observed during and after acceleration stress. During +6 Gz with the anti-G suit inflated, arterial pressure and dP/dt were maintained whereas left ventricular end-diastolic pressure and total peripheral resistance were much elevated and heart rate was lower. At the onset of G stress, internal diameter of the heart always fell transiently. Otherwise, diameter was not significantly affected by any of the experimental conditions. The results suggest that the anti-G suit maintains perfusion pressure at high sustained G; however, with the anti-G suit inflated at +6 Gz, central venous pressure is dramatically elevated and heart rate depressed. Thus, beneficial effects which provide tolerance to high G are accompanied by potentially detrimental effects.
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Peterson et al. (1977) studied this question.
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